CHAPTER
15
THE CHROMOSOMAL BASIS OF INHERITANCE
1. Mendelian inheritance has its physical basis in
the behavior of chromosomes during sexual life cycles
·
Around
1900, cytologists and geneticists began to see parallels between the behavior
of chromosomes and the behavior of Mendel’s factors.
·
Chromosomes
and genes are both present in pairs in diploid cells.
·
Homologous
chromosomes separate and alleles segregate during meiosis.
·
Fertilization
restores the paired condition for both chromosomes and genes.
·
Around
1902, Walter Sutton, Theodor Boveri, and others noted these parallels and a chromosome theory of inheritance began
to take form.
2. Morgan
traced a gene to a specific chromosome
·
Thomas
Hunt Morgan was the first to associate a specific gene with a specific
chromosome in the early 20th century.
·
Like
Mendel, Morgan made an insightful choice as an experimental animal, Drosophila melanogaster, a fruit fly
species that eats fungi on fruit.
·
Fruit
flies are prolific breeders and have a generation time of two weeks.
·
Fruit
flies have three pairs of autosomes and a pair of sex chromosomes (XX in
females, XY in males).
·
Morgan
spent a year looking for variant individuals among the flies he was breeding.
·
He
discovered a single male fly with white eyes instead of the usual red.
·
The
normal character phenotype is the wild
type.
·
Alternative
traits are mutant phenotypes.
·
When
Morgan crossed his white-eyed male with a red-eyed female, all the F1
offspring had red eyes,
·
The
red allele appeared dominant to the white allele.
·
Crosses
between the F1 offspring produced the classic 3:1 phenotypic ratio
in the F2 offspring.
·
Surprisingly,
the white-eyed trait appeared only in males.
·
All
the females and half the males had red eyes.
·
Morgan
concluded that a fly’s eye color was linked to its sex.
·
Morgan
deduced that the gene with the white-eyed mutation is on the X chromosome
alone, a sex-linked gene.
·
Females
(XX) may have two red-eyed alleles and have red eyes or may be heterozygous and
have red eyes.
3. Linked genes tend to be inherited together
because they are located on the same chromosome
4. Independent assortment of chromosomes and
crossing over produce genetic recombinants
5. Geneticists can use recombination data to map a
chromosome’s genetic loci
B.
Sex Chromosomes
1.
The chromosomal basis of sex varies with the organism
·
Although the anatomical and physiological differences between
women and men are numerous, the chromosomal basis of sex is rather simple.
·
In human and other mammals, there are two varieties of sex
chromosomes, X and Y.
·
An individual who inherits two X chromosomes usually develops as a
female.
·
An individual who inherits an X and a Y chromosome usually
develops as a male.
·
This X-Y system of mammals is not the only chromosomal mechanism
of determining sex.
·
Other options include the X-0 system, the Z-W system, and the
haplo-diploid system.
·
In the X-Y system, Y and X chromosomes behave as homologous
chromosomes during meiosis.
·
In reality, they are only partially homologous and rarely undergo
crossing over.
·
In both testes (XY) and ovaries (XX), the two sex chromosomes
segregate during meiosis and each gamete receives one.
·
Each egg receives an X chromosome.
·
Half the sperm receive an X chromosome and half receive a Y
chromosome.
·
Because of this, each conception has about a fifty-fifty chance of
producing a particular sex.
·
In humans, the anatomical signs of sex first appear when the
embryo is about two months old.
·
In individuals with the SRY
gene (sex-determining region of the Y chromosome), the generic embryonic gonads
are modified into testes.
·
Activity of the SRY gene
triggers a cascade of biochemical, physiological, and anatomical features
because it regulates many other genes.
·
In addition, other genes on the Y chromosome are necessary for the
production of functional sperm.
·
In individuals lacking the SRY
gene, the generic embryonic gonads develop into ovaries.
2.
Sex-linked genes have unique patterns of inheritance
·
In addition to their role in determining sex, the sex chromosomes,
especially the X chromosome, have genes for many characters unrelated to sex.
·
These sex-linked genes
follow the same pattern of inheritance as the white-eye locus in Drosophila.
·
If a sex-linked trait is due to a recessive allele, a female will
have this phenotype only if homozygous.
·
Heterozygous females will be carriers.
·
Because males have only one X chromosome (hemizygous), any male receiving the recessive allele from his
mother will express the trait.
·
The chance of a female inheriting a double dose of the mutant
allele is much less than the chance of a male inheriting a single dose.
·
Therefore, males are far more likely to inherit sex-linked
recessive disorders than are females.
·
Several serious human disorders are sex-linked.
·
Duchenne
muscular dystrophy affects one in 3,500 males born in the United States.
·
Affected individuals rarely live past their early 20s.
·
This disorder is due to the absence of an X-linked gene for a key
muscle protein, called dystrophin.
·
The disease is characterized by a progressive weakening of the
muscles and a loss of coordination.
·
Hemophilia is a sex-linked
recessive trait defined by the absence of one or more clotting factors.
·
These proteins normally slow and then stop bleeding.
·
Individuals with hemophilia have prolonged bleeding because a firm
clot forms slowly.
·
Bleeding in muscles and joints can be painful and lead to serious
damage.
·
Individuals can be treated with intravenous injections of the
missing protein.
·
Sex-linked
traits are not the only notable deviation from the inheritance patterns
observed by Mendel.
·
Also,
gene mutations are not the only kind of changes to the genome that can affect
phenotype.
·
Major
chromosomal aberrations and their consequences produce exceptions to standard
chromosome theory.
·
In
addition, two types of normal inheritance also deviate from the standard
pattern.
1. Alterations of chromosome number or structure
cause some genetic disorders
·
Nondisjunction occurs when problems with
the meiotic spindle cause errors in daughter cells.
·
This
may occur if tetrad chromosomes do not separate properly during meiosis I.
·
Alternatively,
sister chromatids may fail to separate during meiosis II.
·
As
a consequence of nondisjunction, some gametes receive two of the same type of
chromosome and another gamete receives no copy.
·
Offspring
results from fertilization of a normal gamete with one after nondisjunction
will have an abnormal chromosome number or aneuploidy.
·
Trisomic cells have three copies of
a particular chromosome type and have 2n
+ 1 total chromosomes.
·
Monosomic cells have only one copy of
a particular chromosome type and have 2n
- 1 chromosomes.
·
If
the organism survives, aneuploidy typically leads to a distinct phenotype.
·
Aneuploidy
can also occur during failures of the mitotic spindle.
·
If
aneuploidy happens early in development, this condition will be passed along by
mitosis to a large number of cells.
·
This
is likely to have a substantial effect on the organism.
·
Organisms
with more than two complete sets of chromosomes, have undergone polypoidy.
·
This
may occur when a normal gamete fertilizes another gamete in which there has
been nondisjunction of all its chromosomes.
·
The
resulting zygote would be triploid (3n).
·
Alternatively,
if a 2n zygote failed to divide after
replicating its chromosomes, a tetraploid
(4n) embryo would result from
subsequent successful cycles of mitosis.
·
Polyploidy
is relatively common among plants and much less common among animals.
·
Breakage
of a chromosome can lead to four types of changes in chromosome structure.
·
A
deletion occurs when a chromosome
fragment lacking a centromere is lost during cell division.
·
This
chromosome will be missing certain genes.
·
A
duplication occurs when a fragment
becomes attached as an extra segment to a sister chromatid.
·
An inversion occurs when a chromosomal
fragment reattaches to the original chromosome but in the reverse orientation.
·
In
translocation, a chromosomal
fragment joins a nonhomologous chromosome.
·
Some
translocations are reciprocal, others are not.
·
Certain
aneuploid conditions upset the balance less, leading to survival to birth and
beyond.
·
These
individuals have a set of symptoms — a syndrome — characteristic of the type of
aneuploidy.
·
One
aneuploid condition, Down syndrome,
is due to three copies of chromosome 21.
·
It
affects one in 700 children born in the United States.
·
Although
chromosome 21 is the smallest human chromosome, it severely alters an
individual’s phenotype in specific ways.
·
Most
cases of Down syndrome result from nondisjunction during gamete production in
one parent.
·
The
frequency of Down syndrome correlates with the age of the mother.
·
This
may be linked to some age-dependent abnormality in the spindle checkpoint
during meiosis I, leading to nondisjunction.
·
Trisomies
of other chromosomes also increase in incidence with maternal age, but it is
rare for infants with these autosomal trisomies to survive for long.
·
Nondisjunction
of sex chromosomes produces a variety of aneuploid conditions in humans.
·
Unlike
autosomes, this aneuploidy upsets the genetic balance less severely.
·
This
may be because the Y chromosome contains relatively few genes.
·
Also,
extra copies of the X chromosome become inactivated as Barr bodies in somatic
cells.
·
Klinefelter’s syndrome, an XXY male, occurs once
in every 2000 live births.
·
These
individuals have male sex organs, but are sterile.
·
There
may be feminine characteristics.
·
Their
intelligence is normal.
·
Males
with an extra Y chromosome (XYY) tend to somewhat taller than average.
·
Trisomy
X (XXX), which occurs once in every 2000 live births, produces healthy females.
·
Monosomy
X or Turner’s syndrome (X0), which
occurs once in every 5000 births, produces phenotypic, but immature females.
·
Some
individuals with Down syndrome have the normal number of chromosomes but have
all or part of a third chromosome 21 attached to another chromosome by
translocation.
2. The phenotypic effects of some mammalian genes
depend on whether they were inherited from the mother or the father
(imprinting)
3. Extranuclear genes exhibit a non-Mendelian
pattern of inheritance
·
Not
all of a eukaryote cell’s genes are located in the nucleus.
·
Extranuclear
genes are found on small circles of DNA in mitochondria and chloroplasts.
·
These
organelles reproduce themselves.
·
Their
cytoplasmic genes do not display Mendelian inheritance.
·
They
are not distributed to offspring during meiosis.